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Full text of this paper is not available in the ETD Center. Copies may be available for inter-library loan from University of Cincinnati or may be available for purchase from Proquest/UMI

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HEALTH PROMOTION AND PRESYMPTOMATIC GENETIC TESTING

FOLEY, SUSAN MARIE
http://rave.ohiolink.edu/etdc/view?acc_num=ucin1005250853

Abstract Details

2001, PhD, University of Cincinnati, Nursing : Adult Health Nursing.
Presymptomatic genetic testing (PGT) makes it possible to predict the onset of diseases that may occur later in life. PGT may also, in the case of multifactorial illness, have an impact on motivation to change behavior and therefore, personal health. The purposes of this study were (a) to explore the influence of engagement in health promoting behaviors (HPB) on the decision to have presymptomatic genetic testing (PGT) for selected adult- onset multifactorial disease and (b) to identify the influence of hypothetical-positive genetic test results on HPB. Pender's Health Promotion Model provided the theoretical framework and the definition for HPB (a multidimensional pattern of self-initiated actions and perceptions that serve to maintain or enhance wellness). The study used a descriptive-comparative design and a convenience sample of 294 individuals living in one Ohio community. Subjects completed a survey in two parts (A and B). Part A contained two data collection instruments: Health Promoting Lifestyle Profile II (HPLPII) to measure HPB and an investigator-developed questionnaire to measure the decision to have PGT. For Part B, subjects were asked to consider that they had received a hypothetical-positive PGT result for one of four pre-selected diseases (heart disease, diabetes, depression, prostate/ovarian cancer) and again complete the HPLPII. Subjects were stratified into either a low (n=128) or high (n=150) HPB group based on HPLPII scores from form A. The majority of subjects (94%) demonstrated an interest in PGT regardless of whether they had high or low HPB (t=-1.096, df=276, n.s). Subjects were less willing to engage in PGT for depression (Cochran's Q = 52.382, df=9, p<.05) when compared to other multifactorial diseases (diabetes, heart disease, high blood pressure, colon, breast, prostate, and ovarian cancers, glaucoma, stroke, and high cholesterol). Finally, a significant increase in HPB from baseline (F=396.662, df=3, p<.05) was found upon introduction of a hypothetical-positive PGT result. For PGT to be a successful tool in healthcare, it must be valued and viewed as useful by the people for whom it is intended. Subjects showed interest not only in having PGT but also indicated they would change HPB when confronted with positive test results.
Dr. Marilyn S. Sommers (Advisor)
1 p.
HEALTH PROMOTION MODEL; GENETIC TESTING

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Citations

  • FOLEY, S. M. (2001). HEALTH PROMOTION AND PRESYMPTOMATIC GENETIC TESTING [Doctoral dissertation, University of Cincinnati]. OhioLINK Electronic Theses and Dissertations Center. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1005250853

    APA Style (7th edition)

  • FOLEY, SUSAN. HEALTH PROMOTION AND PRESYMPTOMATIC GENETIC TESTING. 2001. University of Cincinnati, Doctoral dissertation. OhioLINK Electronic Theses and Dissertations Center, http://rave.ohiolink.edu/etdc/view?acc_num=ucin1005250853.

    MLA Style (8th edition)

  • FOLEY, SUSAN. "HEALTH PROMOTION AND PRESYMPTOMATIC GENETIC TESTING." Doctoral dissertation, University of Cincinnati, 2001. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1005250853

    Chicago Manual of Style (17th edition)

ucin1005250853
© 2001, all rights reserved.
This open access ETD is published by University of Cincinnati and OhioLINK.